The main characteristics of ventricular diastolic filling is blocked. Cause The disease is idiopathic. Pathological Cardiac fibrosis hyperplasia; heart endometrial hyperplasia fiber hypertrophy; ventricular endometrial sclerosis, limited expansion. Clinical performance [Symptoms] Eosinophils by the number of cells caused by myocardial fibrosis in multiple men (male to female ratio of 3:1) Most of the age of 15-50 years old. According to ventricular been involved on both sides of the degree of clinical type can be divided into the right ventricle, left ventricle and mixed-type. The left ventricle of the most common. In the early stages fibrosis, patients can be asymptomatic or because of minor symptoms and can not be identified, with the condition can often progress in burnout, fatigue, labor and difficulty in breathing, after these symptoms increase, there can be chest pain. Among them, there can be early left ventricular-left ventricular dysfunction performance, such as easy fatigue, difficulty in breathing, coughing and lung, such as wet-Lo. The right ventricle and mixed-often consisting mainly of right heart dysfunction. [Signs] There may be signs of right heart failure, such as the jugular vein Nuzhang and suction pressure increased when the jugular vein, liver, ascites, lower limb or body, such as edema, cardiac auscultation and the third heart sound can be heard Benma law. When the mitral valve or tricuspid involvement at the apex or tricuspid regurgitation of the area in systolic murmur, atrial fibrillation, a thromboembolism, is not uncommon. In addition, regular low blood pressure, pulse pressure to narrow. Even in cases of serious heart can be sudden death. Check ECG: This shows that a variety of cardiac anomalies, to atrial fibrillation, T-Diping or inverted. Diagnosis The diagnosis of the disease more difficult, with performance-fatigue, difficulty in breathing, ascites, such as edema around performance similar to cardiac tamponade, the heart is normal or mild increase, auscultation a third heart sound or fourth heart sound, or a slight contraction apex District Phase noise, should consider the disease, such as the original number of eosinophils have a history (fever, cough, flustered, blood eosinophil number, etc.), but also help diagnosis. Combination of laboratory tests, such as electrocardiogram showed ventricular hypertrophy, atrial increased resistance to-conduction band, echocardiography icon endometrial thickening and heart apex occlusion, the diagnosis can be established. Treatment and prognosis The disease prognosis is poor, only symptomatic treatment. Including mitigation of the main principles of clinical symptoms, improve diastolic heart function, to correct heart failure; against the onset of treatment and surgery for serious cases.
